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Kreye, Jakob, Wright, Sukhvir K, van Casteren, Adriana, Stöffler, Laura, Machule, Marie-Luise, Reincke, S Momsen, Nikolaus, Marc, van Hoof, Scott, Sanchez-Sendin, Elisa, Homeyer, Marie A, Cordero Gómez, César, Kornau, Hans-Christian, Schmitz, Dietmar, Kaindl, Angela M, Boehm-Sturm, Philipp, Mueller, Susanne, Wilson, Max A, Upadhya, Manoj A, Dhangar, Divya R, Greenhill, Stuart, Woodhall, Gavin, Turko, Paul, Vida, Imre, Garner, Craig C, Wickel, Jonathan, Geis, Christian, Fukata, Yuko, Fukata, Masaki and Prüss, Harald (2021). Encephalitis patient-derived monoclonal GABAA receptor antibodies cause epileptic seizures. Journal of Experimental Medicine, 218 (11),

Wright, Sukhvir K, Rosch, Richard E, Wilson, Max A, Upadhya, Manoj A, Dhangar, Divya R, Clarke-Bland, Charlie, Wahid, Tamara T, Barman, Sumanta, Goebels, Norbert, Kreye, Jakob, Prüss, Harald, Jacobson, Leslie, Bassett, Danielle S, Vincent, Angela, Greenhill, Stuart D and Woodhall, Gavin L (2021). Multimodal electrophysiological analyses reveal that reduced synaptic excitatory neurotransmission underlies seizures in a model of NMDAR antibody-mediated encephalitis. Communications Biology, 4 (1),

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