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Yang, Yang, Jayaprakash, Deepika, Jhujh, Satpal S., Reynolds, John J., Chen, Steve, Gao, Yanzhe, Anand, Jay Ramanlal, Mutter-Rottmayer, Elizabeth, Ariel, Pablo, An, Jing, Cheng, Xing, Pearce, Kenneth H., Blanchet, Sophie-Anne, Nandakumar, Nandana, Zhou, Pei, Fradet-Turcotte, Amélie, Stewart, Grant S. and Vaziri, Cyrus (2024). PCNA-binding activity separates RNF168 functions in DNA replication and DNA double-stranded break signaling. Nucleic Acids Research, 52 (21),

Krishnan, Rehna, Lapierre, Mariah, Gautreau, Brandon, Nixon, Kevin C.J., El Ghamrasni, Samah, Patel, Parasvi S., Hao, Jun, Yerlici, V. Talya, Guturi, Kiran Kumar Naidu, St-Germain, Jonathan, Mateo, Francesca, Saad, Amine, Algouneh, Arash, Earnshaw, Rebecca, Shili, Duan, Seitova, Alma, Miller, Joshua, Khosraviani, Negin, Penn, Adam, Ho, Brandon, Sanchez, Otto, Hande, M. Prakash, Masson, Jean Yves, Brown, Grant W., Alaoui-Jamali, Moulay, Reynolds, John J., Arrowsmith, Cheryl, Raught, Brian, Pujana, Miguel A., Mekhail, Karim, Stewart, Grant S., Hakem, Anne and Hakem, Razqallah (2023). RNF8 ubiquitylation of XRN2 facilitates R-loop resolution and restrains genomic instability in BRCA1 mutant cells. Nucleic Acids Research, 51 (19), pp. 10484-10505.

Patel, Parasvi S, Algouneh, Arash, Krishnan, Rehna, Reynolds, John J, Nixon, Kevin C J, Hao, Jun, Lee, Jihoon, Feng, Yue, Fozil, Chehronai, Stanic, Mia, Yerlici, Talya, Su, Peiran, Soares, Fraser, Liedtke, Elisabeth, Prive, Gil, Baider, Gary D, Pujana, Miquel Angel, Mekhail, Karim, He, Housheng Hansen, Hakem, Anne, Stewart, Grant S and Hakem, Razqallah (2023). Excessive transcription-replication conflicts are a vulnerability of BRCA1-mutant cancers. Nucleic Acids Research, 51 (9), pp. 4341-4362.

Grange, Laura J, Reynolds, John J, Ullah, Farid, Isidor, Bertrand, Shearer, Robert F, Latypova, Xenia, Baxley, Ryan M, Oliver, Antony W, Ganesh, Anil, Cooke, Sophie L, Jhujh, Satpal S, McNee, Gavin S, Hollingworth, Robert, Higgs, Martin R, Natsume, Toyoaki, Khan, Tahir, Martos-Moreno, Gabriel Á., Chupp, Sharon, Mathew, Christopher G., Parry, David, Simpson, Michael A., Nahavandi, Nahid, Yüksel, Zafer, Drasdo, Mojgan, Kron, Anja, Vogt, Petra, Jonasson, Annemarie, Seth, Saad Ahmed, Gonzaga-Jauregui, Claudia, Brigatti, Karlla W, Stegmann, Alexander P A, Kanemaki, Masato, Josifova, Dragana, Uchiyama, Yuri, Oh, Yukiko, Morimoto, Akira, Osaka, Hitoshi, Ammous, Zineb, Argente, Jesús, Matsumoto, Naomichi, Stumpel, Constance T.R.M., Taylor, Alexander M.R., Jackson, Andrew P., Bielinsky, Anja-Katrin, Mailand, Niels, Le Caignec, Cedric, Davis, Erica E. and Stewart, Grant S. (2022). Pathogenic variants in SLF2 and SMC5 cause segmented chromosomes and mosaic variegated hyperploidy. Nature Communications, 13 (1),

Abu-Libdeh, Bassam, Jhujh, Satpal S, Dhar, Srijita, Sommers, Joshua A, Datta, Arindam, Longo, Gabriel M C, Grange, Laura J, Reynolds, John J, Cooke, Sophie L, McNee, Gavin S, Hollingworth, Robert, Woodward, Beth L, Ganesh, Anil N, Smerdon, Stephen J, Nicolae, Claudia M, Durlacher-Betzer, Karina, Molho-Pessach, Vered, Abu-Libdeh, Abdulsalam, Meiner, Vardiella, Moldovan, George-Lucian, Roukos, Vassilis, Harel, Tamar, Brosh, Robert M and Stewart, Grant S (2022). RECON syndrome is a genome instability disorder caused by mutations in the DNA helicase RECQL1. Journal of Clinical Investigation, 132 (5),

Zhang, Jing, Bellani, Marina A, James, Ryan C, Pokharel, Durga, Zhang, Yongqing, Reynolds, John J, McNee, Gavin S, Jackson, Andrew P, Stewart, Grant S and Seidman, Michael M (2020). DONSON and FANCM associate with different replisomes distinguished by replication timing and chromatin domain. Nature Communications, 11 (1),

Kalasova, Ilona, Hanzlikova, Hana, Gupta, Neerja, Li, Yun, Altmüller, Janine, Reynolds, John J, Stewart, Grant S, Wollnik, Bernd, Yigit, Gökhan and Caldecott, Keith W (2019). Novel PNKP mutations causing defective DNA strand break repair and PARP1 hyperactivity in MCSZ. Neurology Genetics, 5 (2),