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van Hoof, Scott, Kreye, Jakob, Cordero-Gómez, César, Hoffman, Julius, Momsen Reincke, S., Sanchez-Sendin, Elisa, Duong, Sophie L., Upadhya, Manoj, Dhangar, Divya, Michór, Paulina, Woodhall, Gavin L., Küpper, Maraike, Oder, Andreas, Kuchling, Joseph, Paul Koch, Stefan, Mueller, Susanne, Boehm-Sturm, Philipp, Peter von Kries, Jens, Finke, Carsten, Kirschstein, Timo, Wright, Sukhvir K. and Prüss, Harald (2024). Human cerebrospinal fluid monoclonal CASPR2 autoantibodies induce changes in electrophysiology, functional MRI, and behavior in rodent models. Brain, Behavior, and Immunity, 122 , pp. 266-278.

Upadhya, Manoj, Kirmann, Toni, Wilson, Max A., Simon, Christian M., Dhangar, Divya, Geis, Christian, Williams, Robyn, Woodhall, Gavin, Hallermann, Stefan, Irani, Sarosh R. and Wright, Sukhvir K. (2024). Peripherally-derived LGI1-reactive monoclonal antibodies cause epileptic seizures in vivo. Brain, 147 (8), pp. 2636-2642.

Kreye, Jakob, Wright, Sukhvir K, van Casteren, Adriana, Stöffler, Laura, Machule, Marie-Luise, Reincke, S Momsen, Nikolaus, Marc, van Hoof, Scott, Sanchez-Sendin, Elisa, Homeyer, Marie A, Cordero Gómez, César, Kornau, Hans-Christian, Schmitz, Dietmar, Kaindl, Angela M, Boehm-Sturm, Philipp, Mueller, Susanne, Wilson, Max A, Upadhya, Manoj A, Dhangar, Divya R, Greenhill, Stuart, Woodhall, Gavin, Turko, Paul, Vida, Imre, Garner, Craig C, Wickel, Jonathan, Geis, Christian, Fukata, Yuko, Fukata, Masaki and Prüss, Harald (2021). Encephalitis patient-derived monoclonal GABAA receptor antibodies cause epileptic seizures. Journal of Experimental Medicine, 218 (11),

Wright, Sukhvir K, Rosch, Richard E, Wilson, Max A, Upadhya, Manoj A, Dhangar, Divya R, Clarke-Bland, Charlie, Wahid, Tamara T, Barman, Sumanta, Goebels, Norbert, Kreye, Jakob, Prüss, Harald, Jacobson, Leslie, Bassett, Danielle S, Vincent, Angela, Greenhill, Stuart D and Woodhall, Gavin L (2021). Multimodal electrophysiological analyses reveal that reduced synaptic excitatory neurotransmission underlies seizures in a model of NMDAR antibody-mediated encephalitis. Communications Biology, 4 (1),