Nitric oxide-dependent bone marrow progenitor mobilization by carbon monoxide enhances endothelial repair after vascular injury


Carbon monoxide (CO) has emerged as a vascular homeostatic molecule that prevents balloon angioplasty-induced stenosis via antiproliferative effects on vascular smooth muscle cells. The effects of CO on reendothelialization have not been evaluated.

Publication DOI:
Divisions: College of Health & Life Sciences > School of Biosciences
College of Health & Life Sciences > School of Biosciences > Cellular and Molecular Biomedicine
College of Health & Life Sciences > Aston Medical School
College of Health & Life Sciences
Additional Information: © 2010 American Heart Association, Inc.
Uncontrolled Keywords: angioplasty,endothelium,nitric oxide synthase,signal transduction,stenosis,Physiology (medical),Cardiology and Cardiovascular Medicine
Publication ISSN: 1524-4539
Last Modified: 14 Jun 2024 07:10
Date Deposited: 06 Oct 2014 10:55
Full Text Link:
Related URLs: http://www.scop ... tnerID=8YFLogxK (Scopus URL)
http://circ.aha ... ntent/121/4/537 (Publisher URL)
PURE Output Type: Article
Published Date: 2010-02
Authors: Wegiel, Barbara
Gallo, David J.
Raman, Kathleen G.
Karlsson, Jenny M.
Ozanich, Brett
Chin, Beek Y.
Tzeng, Edith
Ahmad, Shakil (ORCID Profile 0000-0002-9294-0475)
Ahmed, Asif (ORCID Profile 0000-0002-8755-8546)
Baty, Catherine J.
Otterbein, Leo E.



Version: Accepted Version

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