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2015
Houée-Lévin, C.; Bobrowski, K.; Horakova, L.; Karademir, B.; Schöneich, C.; Davies, M.J. and Spickett, C.M. (2015). Exploring oxidative modifications of tyrosine:an update on mechanisms of formation, advances in analysis and biological consequences. Free Radical Research, 49 (4), pp. 347-373.
2013
Spickett, C.M.; Tveen-jensen, K.; Reis, A. and Pitt, A.R. (2013). Protein modification and phospholipid oxidation. Free Radical Biology and Medicine, 65 (Suppl.), S15.
2012
Reis, A.; Spickett, C.M. and Rudnitskaya, A. (2012). Integrated Omics for the global mapping of biomolecules in LDL. Free Radical Biology and Medicine, 53 (Supple), S212.
Pasha, S.; Mahalka, A.K.; Reis, A.; Pitt, A.R.; Kinnunen, P.K.J. and Spickett, C.M. (2012). Mass spectrometry analysis of modified bee venom phospholipase A2 in correlation with increased activity. Free Radical Biology and Medicine, 53 (Supple), S213-S214.
2009
Strosova, M.; Karlovska, J.; Spickett, C.M. and Horakova, L. (2009). Mechanism of serca modulation from rats with adjuvant arthritis. IN: SFRR Europe Meeting 2009 Free radicals, Health and Lifestyle: from cell signalling to disease prevention. 2009-09-27.
2008
Spickett, C.M. (2008). Chlorinated and oxidized lipids in inflammation. IN: Meeting of the Society-for-Free-Radical-Research-Europe. 2009-07-05 - 2009-07-09.
Taylor, Michelle K.; Trotter, Katherine D.; Reglinski, John; Berlouis, Leonard E.A.; Kennedy, Alan R.; Spickett, C.M. and Sowden, Rebecca J. (2008). Copper N2S2 Schiff base macrocycles:the effect of structure on redox potential. Inorganica Chimica Acta, 361 (9-10), pp. 2851-2862.
2007
Mouls, L.; Silajdzic, E.; Pitt, A.R. and Spickett, C.M. (2007). Development of novel mass spectrometry methodology to detect post-translational modifications in oxidative stress and disease. Free Radical Research, 41 , S43.
Dever, G.; Spickett, C.M.; Kennedy, S.; Rush, C.; Tennant, G.; Monopoli, A. and Wainwright, C.L. (2007). The nitric oxide-donating pravastatin derivative, NCX 6550 [(1S-[1α(βS*, δS*), 2α, 6α, 8β-(R*), 8aα]]-1,2,6,7,8,8a-hexahydro-β, δ, 6-trihydroxy-2-methyl-8-(2-methyl-1-oxobutoxy)-1-naphtalene-heptanoic acid 4-(nitrooxy)butyl ester)], reduces splenocyte adhesion and reactive oxygen species generation in normal and atherosclerotic mice. Journal of Pharmacology and Experimental Therapeutics, 320 (1), pp. 419-426.
2005
Spickett, C.M.; Dever, G.; Kennedy, S. and Wainwright, C.L. (2005). Studies of potential atherogenic properties of phospholipid chlorohydrins. Free Radical Research, 39 (Suppl.), S79.
2003
Dever, G.; Oliveira, C.P.; Stewart, L.J. and Spickett, C.M. (2003). The biological effects of chlorohydrins and other products of HOCL attack:relevance to inflammation. Free Radical Research, 37 , p. 27.